AI viva examiner

A viva examiner that asks the next question

Not a question bank. An examiner that listens to your answer, marks it on six domains, tells you what it thought, and asks the follow-up a real examiner would ask. At the end you get the report they would write.

  • Your subject and topic, at undergraduate or postgraduate level and the difficulty you choose
  • Every answer marked 0 to 10 on accuracy, understanding, reasoning, application, completeness and communication
  • A closing report: strengths, weaknesses, missed points and a revision plan

Included in

  • Student Pro10 a day
  • Faculty Pro20 a day
  • InstitutionUnlimited

Not in the free tier, but every new account starts with a 7-day Student Pro trial, no card, so you can use it from day one.

How a session works

  1. 1

    Choose the viva

    Subject, topic, level and difficulty, and how many questions you want. Pharmacology, medicine, or a topic from your own syllabus taxonomy.

  2. 2

    Answer in your own words

    Type your answer as you would say it. The examiner reads it as evidence, marks it, and gives two or three sentences of feedback.

  3. 3

    Take the follow-up

    The next question depends on your answer: it probes a gap, presses a misconception, or moves on if you were complete.

  4. 4

    Read the report

    An overall score, strengths, weaknesses, the points you missed, and a revision plan written for you.

Try it now, no account

Read a full session

The examiner cannot be opened without an account, because every question costs a model call. This is what a session produces: an abridged four-question viva on opioid analgesics, with the marks, the feedback and the closing report in the examiner's own format.

PharmacologyOpioid analgesicsUndergraduateMedium

Examiner, question 1

Let us start with morphine. Tell me how it produces analgesia, and where in the nervous system that happens.

Candidate

Morphine is a full agonist at mu opioid receptors. They are G protein coupled receptors, Gi linked, so activation reduces cAMP, opens potassium channels which hyperpolarises the neuron, and closes voltage gated calcium channels which reduces transmitter release. It acts at several levels: in the dorsal horn it reduces release of substance P and glutamate from primary afferents, in the periaqueductal grey it switches on descending inhibition, and supraspinally it changes the emotional response to pain.

Marks

8.3 / 10

Accuracy
9
Understanding
9
Reasoning
8
Application
7
Completeness
8
Communication
9

Feedback. A clear, well organised answer that moves from receptor to circuit. You could have added the role of peripheral opioid receptors in inflamed tissue, but the core mechanism was complete and correctly stated.

Examiner, question 2

Good. Now the other side. A patient on regular morphine becomes drowsy with a respiratory rate of 8. Walk me through what is happening and what else you would expect to see.

Candidate

That is respiratory depression. Morphine reduces the sensitivity of the brainstem respiratory centres to carbon dioxide, so the drive to breathe falls. I would expect pinpoint pupils, and probably reduced consciousness. Other effects would be constipation, nausea, and itching.

Marks

6.5 / 10

Accuracy
8
Understanding
7
Reasoning
6
Application
6
Completeness
5
Communication
7

Feedback. Correct on the mechanism and the pupils. The question asked you to reason about this patient: why now, after being stable on regular morphine? You listed adverse effects instead of asking what had changed, which is the clinical question.

Examiner, question 3

Exactly the point I want to press. Her creatinine has doubled over the week. Why does that matter for morphine specifically?

Candidate

Because morphine is cleared by the kidneys, so if renal function falls the drug accumulates and its effect is prolonged. The dose would need reducing.

Marks

4.8 / 10

Accuracy
5
Understanding
4
Reasoning
5
Application
5
Completeness
3
Communication
7

Feedback. Half right, and the missing half is the important one. Morphine itself is cleared mainly by hepatic glucuronidation. What accumulates in renal failure are its glucuronide metabolites, and morphine-6-glucuronide is a potent opioid agonist in its own right. That is why the patient deteriorates days into a stable regimen, and why the usual advice is to switch to an opioid without active renal metabolites rather than only to reduce the dose.

Misconceptions noted: Morphine is eliminated unchanged by the kidney; Renal impairment is handled by dose reduction alone

Examiner, question 4

Last one. She is now unresponsive with a rate of 6. What do you do, and what do you warn the nurses about afterwards?

Candidate

Airway, breathing, circulation first, with oxygen and bag valve mask ventilation if she is not breathing adequately. Then naloxone, a mu receptor antagonist, given IV in small titrated doses, 100 to 200 micrograms at a time, aiming to restore breathing rather than full reversal so that we do not precipitate severe pain or withdrawal. The warning is that naloxone has a much shorter half life than morphine, so she can re-sedate, and here the active metabolites will hang around for days. She needs close monitoring and may need repeated doses or an infusion. I would also stop the morphine and involve the pain team about an alternative.

Marks

9.0 / 10

Accuracy
9
Understanding
9
Reasoning
9
Application
9
Completeness
9
Communication
9

Feedback. Excellent. Resuscitation before pharmacology, titrated rather than bolus reversal with the reason for it, and you tied the re-sedation warning back to the metabolite point from the previous question. That is exactly the integration a viva rewards.

Closing report

Overall 73.3%
x

A candidate with a strong grasp of receptor pharmacology and emergency management, who was caught out on the one question that required pharmacokinetic reasoning about a changing patient. The final answer showed the ability to integrate is there; it needs to be applied earlier and unprompted.

Strengths

  • Mechanism of opioid analgesia explained from receptor to circuit
  • Safe, staged management of opioid toxicity with titrated naloxone
  • Communicates clearly and structures answers well

Weaknesses

  • Reaches for lists of adverse effects when a question asks for reasoning about a specific patient
  • Pharmacokinetics of morphine metabolites not known

Missed points

  • Morphine-6-glucuronide accumulates in renal impairment and is an active agonist
  • Switching opioid, not only reducing the dose, in worsening renal function
  • Peripheral opioid receptors in inflamed tissue

Revision plan

  • Morphine metabolism: glucuronidation, M3G and M6G, and how each behaves in renal failure
  • Opioid choice in renal and hepatic impairment: which agents are safer and why
  • Practise reading a scenario for what has changed before answering

A representative session written for this page, in the format the examiner stores. Your own sessions are saved with every question, answer and mark.

What gets marked

Accuracy

Is what you said correct?

Understanding

Do you know why, not only what?

Reasoning

Can you work from principles to this patient or this question?

Application

Would your answer hold up at the bedside?

Completeness

What did you leave out that an examiner would expect?

Communication

Structure, clarity and confidence, the things that decide a borderline viva.

Questions

Is the examiner an AI?
Yes. It is a language model given an examiner's brief, your chosen topic and level, and a strict marking format. Its marks are stored per question and its report is stored with the session.
Can the examiner be talked into a mark?
No. Answers are quoted to the examiner as evidence rather than instruction, marks must be numbers within range, and an answer that tries to instruct the examiner is marked on its clinical content, which is usually none.
How many questions is a viva?
You choose the target, typically eight. The examiner ends earlier if it has seen enough, or when the target is reached.
Which plan includes it?
The viva examiner is part of Student Pro and above, with a daily allowance of sessions. Every new account starts with a Student Pro trial, no card required.

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